aconditionin which too muchaldosterone(= ahormone)isproducedin theadrenalcortex(=partof thebrain)so that thefluidlevelsin thebodyarewrong,leadingto highbloodpressure
Increased cell production can increase the bulk of an organ, which can causehyperaldosteronism.
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The combination of polydipsia and (nocturnal) polyuria is also seen in (primary)hyperaldosteronism(which often goes with hypokalemia).
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When aldosterone is overproduced, it is calledhyperaldosteronism.
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Adrenal carcinoma is an extremely rare cause of primaryhyperaldosteronism.
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Alcohol stimulates renal excretion of magnesium, which is also increased because of alcoholic and diabetic ketoacidosis, hypophosphatemia andhyperaldosteronismresulting from liver disease.
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Secondaryhyperaldosteronism, on the other hand, is due to overactivity of the renin-angiotensin system.
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A secondaryhyperaldosteronismdevelops due to the hypovolemia.
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The cutoff normal individuals from those with primaryhyperaldosteronismis significantly affected by the conditions of testing, such as posture and time of day.
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This latter sense may rather be termed refractoryhyperaldosteronism.
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Likehyperaldosteronism, it produces hypertension associated with low plasma renin activity, and metabolic alkalosis associated with hypokalemia.
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Primaryhyperaldosteronismare conditions in which the adrenal gland releases too much of the hormone aldosterone.
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His clinic was leading for years after in research onhyperaldosteronism.
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In patients withhyperaldosteronism, chronic exposure to excess aldosterone does not cause edema as might be expected.
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In secondaryhyperaldosteronism, the excess aldosterone is caused by something outside the adrenal gland that mimics the primary condition.
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In addition, patients withhyperaldosteronismwill have hypernatremia, hypokalemia, and metabolic alkalosis.
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